If you were looking for some motivation to follow your doctor's advice or remember to take your medicine, look no further than this grisly tale.
A 64-year-old man went to the emergency department of Brigham and Women’s Hospital in Boston with a painful festering ulcer spreading on his left, very swollen ankle. It was a gruesome sight; the open sore was about 8 by 5 centimeters (about 3 by 2 inches) and was rimmed by black, ashen, and dark purple tissue. Inside, it oozed with streaks and fringes of yellow pus around pink and red inflamed flesh. It was 2 cm deep (nearly an inch). And it smelled.
The man told doctors it had all started two years prior, when dark, itchy lesions appeared in the area on his ankle—the doctors noted that there were multiple patches of these lesions on both his legs. But about five months before his visit to the emergency department, one of the lesions on his left ankle had progressed to an ulcer. It was circular, red, tender, and deep. He sought treatment and was prescribed antibiotics, which he took. But they didn't help.
© Getty | Grace Cary
Food poisoning is never fun—and a lot of the time, it can seem quite forceful. Take the common gut-buster norovirus for example. It can ignite forces that might make jet propulsion researchers jealous. Victims may fear liftoff from a porcelain launch pad, or a vomitous blast with a reverse thrust that seems powerful enough to drop a military jet from the sky.
But then there are the rare illnesses that produce truly violent forces. Such was the case for one unfortunate man in China who made the near-fatal decision to eat some dubious street food.
It's unclear what the 59-year-old ate exactly—but it's a safe bet he'll never eat it again. Soon after, his innards vigorously ignited. According to a case report in this week's issue of the New England Journal of Medicine, the man, in fact, vomited with so much explosive force that he blew apart his esophagus—the muscular tube through which food passes from the throat to the stomach.
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Two years ago, at a Stop & Shop in Rhode Island, the Danish neuroscientist and physician Henriette Edemann-Callesen visited an aisle stocked with sleep aids containing melatonin. She looked around in amazement. Then she took out her phone and snapped a photo to send to colleagues back home.
“It was really pretty astonishing,” she recalled recently.
In Denmark, as in many countries, the hormone melatonin is a prescription drug for treating sleep problems, mostly in adults. Doctors are supposed to prescribe it to children only if they have certain developmental disorders that make it difficult to sleep—and only after the family has tried other methods to address the problem.
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It's well-established that, on the whole, Americans die younger than people in most other high-income countries. For instance, an analysis from 2022 found that the average life expectancy of someone born in Switzerland or Spain in 2019 was 84 years. Meanwhile, the average US life expectancy was 78.8, lower than nearly all other high-income countries, including Canada's, which was 82.3 years. And this was before the pandemic, which only made things worse for the US.
Perhaps some Americans may think that this lower overall life-expectancy doesn't really apply to them if they're middle- or upper-class. After all, wealth inequality and health disparities are huge problems in the US. Those with more money simply have better access to health care and better health outcomes. Well-off Americans live longer, with lifespans on par with their peers in high-income countries, some may think.
It is true that money buys you a longer life in the US. In fact, the link between wealth and mortality may be stronger in the US than in any other high-income country. But, if you think American wealth will put life expectancy in league with Switzerland, you're dead wrong, according to a study in the latest issue of the New England Journal of Medicine.
© Getty | Pascal Mora
A study released on Wednesday finds that a live-virus vaccine that limits shingles symptoms was associated with a drop in the risk for dementia when it was introduced. The work took advantage of the fact that the National Health Service Wales made the vaccine available with a very specific age limit, essentially creating two populations, vaccinated and unvaccinated, separated by a single date. And these populations showed a sharp divide in how often they were diagnosed with dementia, despite having little in the way of other differences in health issues or treatments.
This study didn't come out of nowhere. There have been a number of hints recently that members of the herpesvirus family that can infect nerve cells are associated with dementia. That group includes Varicella zoster, the virus that causes both chicken pox and—potentially many years after— shingles, an extremely painful rash. And over the past couple of years, observational studies have suggested that the vaccine against shingles may have a protective effect.
But it's extremely difficult to do a clinical trial given that the onset of dementia may happen decades after most people first receive the shingles vaccine. That's why the use of NHS Wales data was critical. When the first attenuated virus vaccine for shingles became available, it was offered to a subset of the Welsh population. Those who were born on or after September 2, 1933, were eligible to receive the vaccine. Anyone older than that was permanently ineligible.
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